Monday, August 27, 2012

My Fisher Grad Life blog ? Goodbye CA, Hello OH!

?You left California? for Ohio?!? is the number one question I?ve been asked since arriving in Ohio three short weeks ago. It?s true, California is an amazing place- wonderful weather, beautiful scenery, and of course my incredible family and friends. But Ohio offered me something I couldn?t find in California.

Leaving CA; Entering OH

I graduated from UCLA in June 2011 with a BA in International Development Studies and little intention of going to graduate school (at least not right away). Unfortunately the ?real world? had other plans. I soon realized that a graduate degree and a bit more time to get some relevant career experience could make me a much more desirable employee. I began my search for a graduate program with Ohio State because it is the only university outside of CA to which I had any connection- a good friend attended OSU for undergrad and I visited during Fall 2010. Still, I didn?t want to put all my eggs in one basket so? I applied to three other universities as well.

When decisions rolled in I learned I had been accepted to all four of the universities to which I had applied! But, that meant I had a TOUGH decision ahead of me. I decided to take a trip to three of the four schools to determine which might be the best fit. I met wonderful people and saw exceptional programs in Texas, Indiana, and Ohio. But, as I mentioned, Fisher was where I began my search for a graduate degree program and Ohio State already held a special place in my heart.

My best friend and I after OSU vs Michigan 2010.

The personal connection I already felt intensified during my visit to Fisher. Each person I came in to contact with was so warm and welcoming. It was clear that Fisher is a supportive and collaborative environment- something I intended to find in a graduate program. Throughout the visit Fisher staff, students, and faculty shared their passion for the program and the university with me. They showed me that despite the massive size of Ohio State, at Fisher you are not a number. Fisher cares about each individual and provides them with the education and resources to succeed in their chosen field.

As I prepared to move 2300 miles away from my home and family, I looked forward to finding a new family and home away from home at Fisher. And I am already well on my way. There is no doubt in my mind that leaving CA was a great decision! And hey, I can always go back in two years.

Source: https://fisher.osu.edu/blogs/gradlife/2012/08/27/goodbye-ca-hello-oh/

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Friday, August 10, 2012

ICAST and Uncle Homer's legacy - Florida Sport Fishing | The ...

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Florida Sport Fishing

The International Convention of Allied Sportfishing Trades (ICAST), the largest sport-fishing industry tradeshow in the world, was in Orlando last month.

It was an ideal venue for Florida Fish and Wildlife Conservation Commission (FWC) staff to highlight the 75th anniversary of the Wildlife and Sport Fish Restoration programs and explain how funds from the Sport Fish Restoration (SFR) part of the program enhance Florida?s recreational fishing and boating industries. Those industries respectively generate economic impacts of $8 billion and $17 billion and provide nearly 300,000 Floridians with employment.

Moreover, Florida?s title of the ?Fishing Capital of the World? comes from providing more recreational fishing enjoyment to anglers than any other state and giving them a lifetime of active, nature-based recreation to enhance their physical and mental well-being.

The federal SFR program collects money from manufacturers of fishing equipment and motorboat and small-engine fuels. Those funds, combined with fishing license revenues, help support future fishing and boating opportunities, including FWC programs such as habitat enhancement, fish stocking, building boat ramps and artificial reefs, and youth fishing programs.

Tom Champeau, director of the FWC?s Division of Freshwater Fisheries Management, used a press conference at ICAST to provide an update on TrophyCatch (TrophyCatchFlorida.com). TrophyCatch is the latest federal-state-industry partnership to be partially funded by SFR and largely supported by industry sponsorships and donations to promote catch-and-release of Florida?s trophy largemouth bass.

The TrophyCatch program rewards anglers who catch-and-release largemouth bass greater than 8 pounds. Anglers will be encouraged to follow catch-and-release guidelines for bass weighing 8 to 12.9 pounds and to document the catch with a length, weight and series of photos prior to release. A more thorough certification process will be established regarding Hall-of-Fame bass.

By documenting and publicizing catches of trophy bass, Florida?s bass fisheries will attain even greater prominence. Other catch data helps biologists improve trophy-bass management via habitat enhancement, regulation management, stocking and other proven means that also foster a strong conservation ethic.

TrophyCatch?s corporate partners include the Kissimmee Convention and Visitors Bureau, World Fishing Network, Rapala, FishPhotoReplicas.net, SportsmanOnCanvas.com, Bass Pro Shops, ODU Magazine, Carls Van Rentals, the Recreational Boating and Fishing Foundation, Glen Lau Productions and Under the Bridge Productions. More are joining this support group.

At ICAST, the Professional Outdoor Media Association (POMA) and American Sportfishing Association presented the?Homer Circle Fishing Communicator Award. This award recognizes fishing industry journalists who exemplify the spirit, dedication, talent and commitment to mentoring the next generation of fishing industry communicators that was displayed by Homer Circle during his storied career. The 2012 award went to John E. Phillips, who previously had been inducted into the National Fresh Water Fishing Hall of Fame as a Legendary Communicator.

?Although John?s accomplishments as a communicator are virtually unequalled, his greatest gift to our industry, as is the case with ?Uncle Homer,? is his commitment to mentoring folks, particularly young people, who want to launch a career in the outdoor industry,? said Laurie Lee Dovey of POMA.

This year?s presentation was especially meaningful, since it was the first since Homer Circle died in June. Just five days before he died, even at age 97, he was pursuing his passion by fishing central Florida?s famed bass fisheries.

Known to anglers and would-be anglers alike as Uncle Homer, his renown as a writer began in 1964 when he started selling stories to Sports Afield. His understanding of the piscatorial arts, acumen and prose garnered him the role of angling editor, which he held for 36 years. His column, ?Ask Uncle Homer? in BassMaster Magazine was a classic.

Circle earned ASA?s Lifetime Achievement Award in 1996, and is a member of the Fresh Water Fishing Hall of Fame, the Bass Fishing Hall of Fame and the International Game Fishing Hall of Fame. He hosted ?The Fisherman,? ?Sports Afield? and ?The Outdoorsman,? and was featured in Glen Lau?s classic film ?Bigmouth.? His books included ?The Art of Plug Fishing,? ?New Guide to Bass Fishing,? ?Worming and Plugging for Bass? and ?Circle on Bass and Bass Wisdom.?

His career included a stint as a vice president of Hedon lures. According to Ken Duke and Jeff Samsel (?The Bass Fishing Vault,? 2010), his was one of the most monumentally influential bylines in creating the phenomenon that made black bass the most popular sport fish in the world.

The Fishing Wire (theFishingWire.com) ran an op-ed immediately after Homer?s death that said, ?The outdoor industry, especially those who have ever picked up pens or pencils ? is in mourning after losing a legendary writer, a great friend, and source of inspiration to everyone he met. For decades, Homer Circle has been fishing?s favorite uncle.?

Source: http://floridasportfishing.com/magazine/conservation-corner/florida-fish-and-wildlife-conservation-commission/icast-and-uncle-homer-s-legacy

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Thursday, August 9, 2012

Discover Writing Jobs-India Work Opportunities At Home

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Rob Hayles: We might see a dip in results for GB ... - BBC World News

Three-time Olympic medallist and Metro columnist Rob Hayles sums up an unforgettable Games for British cycling and looks forward to Rio 2016. Jason Kenny (left), Philip Hindes (centre) and Sir Chris Hoy celebrate their team sprint win (Picture: PA) Highs Where to start? It?s been one success story after another for Team GB?s cyclists, with Lizzie Armitstead getting the ball rolling with silver in the women?s road race. Then the velodrome gold rush began with the men?s sprint team of Phillip Hindes, Jason Kenny and Sir Chris Hoy before a sensational victory by Victoria Pendleton in the keirin the same evening. The team pursuit boys did us proud as they destroyed the Australians and the trio of Dani King, Joanna Rowsell and Laura Trott romped to another top podium place in the women?s team pursuit. Laura Trott celebrates the first of her two gold medal runs (Picture: EPA) King Kenny was at it again in the men?s sprint, Jason collecting his second gold on Monday. To top it all, we had little Trotty establishing herself as the new star of the track when she thundered home in the women?s omnium before Chris ended it the only way he knew how by claiming his sixth Olympic title. What more can we say about Sir Chris? Watching him win his sixth gold medal was a real privilege. Lows Jess Varnish and Victoria Pendleton?s disqualification in the team sprint after breaking the world record in qualifying was a blow. As was seeing Mark Cavendish miss out. What now? After Beijing, we saw a dip in results and that might happen again after London but I don?t want to see people say we?ve gone backwards if it does because we will re-focus and keep improving in the build-up to Rio in four years? time. Feet-up time Watching and commentating for the BBC on some of my British team-mates has been fantastic but I have to admit it?s been stressful. The emotions, the highs, the lows ? I feel shattered just thinking about it but I?ve enjoyed every minute. PICTURES: Track cycling on day 11

Source: http://bbc-worldnews.net/2012/08/rob-hayles-we-might-see-a-dip-in-results-for-gb-cyclists-after-london-2012/

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Monday, July 30, 2012

Rare Diseases: 5 Recent Reasons to Cheer

On Sunday morning, July 21, I faced a room of people from families with Leber congenital amaurosis (LCA), an inherited blindness caused by mutations in any of at least 18 genes. It was the final session of the Foundation for Retinal Research?s bi-annual LCA family conference, and I was there to discuss the history of gene therapy. But I zapped through that quickly, because the future is much more intriguing.

Exome sequencing identified the rare mutation that causes Gavin Stevens? hereditary blindness (Leber congenital amaurosis, or LCA). (Troy Stevens)

Exome sequencing identified the rare mutation that causes Gavin Stevens? hereditary blindness (Leber congenital amaurosis, or LCA). (Troy Stevens)

The excitement pervading the room that day was palpable, following a day of scientific updates, and not only because those with young children were soon to visit Sesame World and the sights of Philadelphia.

Jennifer and Troy Stevens exemplified that hope. Two years earlier, at this conference, they?d learned that researchers had been unable to identify a mutation behind their toddler Gavin?s blindness. Now they know the name of their gene: NMNAT1. I?ll return to their story.

The star of the 2010 conference had been 10-year-old Corey Haas and an energetic young sheepdog, both cured of LCA with gene therapy. This weekend, the stars were the new programs and technologies that would allow other families to join Corey?s ? and not just those with blindness.

The rare disease community in the US collectively belies its name: at least 30 million people suffer from 7,000+ diseases, many so rare that they hover beneath the radar of big pharma. But maybe not for long, thanks to the following recent reasons to cheer:

#1: GENE THERAPY PENDING APPROVAL

On July 20, the European Medicines Agency (EMA) announced impending first approval of a gene therapy in the western world.

It?s for lipoprotein lipase deficiency (LPLD). The enzyme normally breaks down tiny triglyceride-packed globules called chylomicrons, and its absence causes episodes of very painful pancreatitis that can be fatal. LPLD is an ultra-rare disease, striking 1-2 people per million. And the only treatment is a diet so low in fat that most patients can?t stick to it.

The gene therapy, Glybera, consists of adeno-associated virus type 1 delivering an overactive variant of the LPL gene, injected into a leg muscle during a single day. But not many people have had it.

James Wilson, MD, PhD, developed the vector, AAV1, used in the lipoprotein lipase deficiency gene therapy. (University of Pennsylvania).

James Wilson, MD, PhD, developed the vector, AAV1, used in the lipoprotein lipase deficiency gene therapy. (University of Pennsylvania).

The research team, led by Daniel Gaudet, MD, PhD, a professor of medicine at the University of Montreal, with colleagues from Amsterdam Molecular Therapeutics (recently replaced by privately-held UniQure), reported a two-year follow-up of 14 adult patients receiving 100 billion to 1 trillion viruses. And it seems to have worked, depending upon how one assesses success.

?The triglycerides dropped, but after 60 days they trended back up. The primary endpoint had failed, but the secondary endpoint was recurring episodes of pancreatitis ? and they found a statistically significant, or close to it, decrease,? explained James Wilson, MD, PhD, editor-in-chief of Human Gene Therapy and professor of pathology and laboratory medicine at the University of Pennsylvania, who developed the vector. Tracking a few more patients, work not yet published, may have led the EMA?s Committee for Medicinal Products for Human Use to finally recommend approval, after three rejections.

Tomas Salmonson, MD, acting chair of the committee, points to the new data as well as restricting use to the sickest patients in pushing the gene therapy forward. ?Our established ways of assessing the benefits and risks of Glybera were challenged by the extreme rarity of the condition and also by uncertainties associated with data provided.?

For the additional study, the researchers looked at what was happening in the chylomicrons in the blood, and found that triglyceride level can fluctuate, contrary to assumptions of steady change. And that means something is happening that might explain the decrease in the painful episodes ? a very real measurement. Summed up Jean Bennett, MD, PhD, leader of one of the LCA2 clinical trials at Penn, ?It?s a huge vote of confidence for the entire field of gene transfer.?

Dr. Wilson agrees. The repercussions won?t be at the FDA, where scientists make decisions based on data, he said, but on the willingness of big pharma to invest in gene therapy. Despite recent successes ? LCA2, hemophilia, adrenoleukodystrophy ? the pharmaceutical industry has been hesitant to fund gene therapy because it has lacked an approval. ?So-called regulatory uncertainty has been the biggest problem, and if there?s no precedent, they can continue to say no. Biopharma is not interested in the ultra orphans. But I have a feeling we?ll be seeing some activity,? he added.

#2: FINDING HOMES FOR STALLED DRUGS

By August 14, researchers can submit pre-applications to the National Center for Advancing Translational Sciences (NCATS) Discovering New Therapeutic Uses for Existing Molecules program. The idea is simple yet brilliant: match compounds that are languishing on company shelves to diseases with newly-discovered mechanisms. Such candidate drugs have passed initial safety tests but were dropped for business reasons, such as a tiny market, or because they didn?t treat what they were intended to.

Corey Haas and Hannah Sames are ambassadors for the rare disease community, here signing their photos in ?The Forever Fix: Gene Therapy and the Boy Who Saved It.? Corey has LCA2, successfully treated with gene therapy, and Hannah, awaiting hers, is one of 54 people in the world who has giant axonal neuropathy. (Sandy Andersen)

Corey Haas and Hannah Sames are ambassadors for the rare disease community, here signing their photos in ?The Forever Fix: Gene Therapy and the Boy Who Saved It.? Corey has LCA2, successfully treated with gene therapy, and Hannah, awaiting hers, is one of 54 people in the world who has giant axonal neuropathy. (Sandy Andersen)

Since the announcement in June, eight industry leaders have signed on, offering an initial 58 compounds to find new therapeutic homes. And the need is compelling: of the 4,500+ diseases with recently-revealed mechanisms, only about 250 have treatments. ?If researchers funded through this effort can demonstrate new uses for the compounds, they could significantly reduce the amount of time it takes to get a treatment to patients in need,? said Kathy L. Hudson, PhD, NCATS acting deputy director.

Everyone wins.

#3: SPEEDING FDA APPROVAL

On July 9, President Obama signed into law the FDA Safety and Innovation Act, which updates the 1983 Orphan Drug Act. The new law provides $6 billion over the next 5 years to assist the agency in evaluating new drugs and medical devices. The Act will speed access to new treatments and development of especially promising ones, and the Humanitarian Use Devices program will target those that treat rare diseases, giving priority to diseases of children. ?Treatments are desperately needed because most are serious, many are life-threatening, and about two-thirds of the patients are children,? said Peter L. Saltonstall, president and CEO of the National Organization for Rare Disorders (NORD), which was critical in developing both acts.

The Act may be a lifesaver for people such as 8-year-old Hannah Sames, one of 54 people in the world known to have giant axonal neuropathy. The gene therapy trial that she will take part in is nearing phase 1, but the sponsoring not-for-profit, Hannah?s Hope Fund, is about to run out of money.

#4: EASING INSURANCE ACCESS

When the Supreme Court upheld the Affordable Care Act on June 28, I scrolled through the relieved statements from various rare disease organizations. Thanks to the ACA, children like Hannah Sames and Gavin Stevens will not be penalized for their pre-existing conditions, nor face annual or lifetime insurance caps.

#5: IDENTIFYING DISEASE GENES

Exome sequencing can identify mutations when single-gene tests don?t. The strategy sequences the protein-encoding part of the human genome in individuals, usually young children, whose syndrome has evaded recognition, searching for mutations passed silently from parents, with functions that could explain the symptoms. Once that?s known, researchers can develop new treatments, or repurpose existing ones.

New exome-derived discoveries are being reported nearly weekly, some appearing in the media before the technical papers are published. A recent news release about a 4-year-old named Maya with a neurological disease, for example, made its way into many news reports and blogs, with a touching story and accolades. Yet none named the gene or its precise function ? the part I?m most interested in.

In contrast to the incomplete Maya story, when John Chiang, PhD, director of the Molecular Diagnostics Laboratory at the Casey Eye Institute in Portland, Oregon told me he?d discovered Gavin Stevens? mutation among nearly 2,500 gene variants in the blind boy?s exome, he asked that I not report it. That was 8 months ago ? the mutation is unveiled in a quartet of papers in the current Nature Genetics, after something of a turf war among four research groups.

Gavin?s parents had heard about Dr. Chiang at the Foundation for Retinal Research meeting two years ago, where Jennifer had called him, distraught, after learning that single-gene tests couldn?t explain their son?s blindness. Dr. Chiang, who described his skill as ?I do the dirty work, I find the mutations,? had helped several families after existing tests had fruitlessly, but expensively, probed the most common parts of only the most common genes. Dr. Chiang had first developed larger gene testing panels, and when those still didn?t identify some families? mutations, quietly sent their DNA off to the Beijing Genome Institute for whole exome sequencing.

Now that exome sequencing is commercially available in the U.S., Dr. Chiang cautions that it still doesn?t help all families, and that costs can greatly exceed the oft-mentioned $1,000 pricetag when considering analysis. ?I would only recommend it as the last resort when all known genes are ruled out,? he advised.

CODA

Karen Poulakos has Leber congenital amaurosis, and does quite well in her world of shadows. Gene therapy may return the vision that she remembers from her childhood. (Ricki Lewis)

Karen Poulakos has Leber congenital amaurosis, and does quite well in her world of shadows. Gene therapy may return the vision that she remembers from her childhood. (Ricki Lewis)

On Saturday at the retinal research conference last weekend, I watched Jennifer and Troy beam as Eric Pierce, MD, PhD, director of the Ocular Genomics Institute in Boston and co-author of one of the Nature Genetics papers, talked about their mutation. Discovery of the gene, which affects cellular energy (NAD synthesis), is a starting point for gene therapy, and this particular candidate is a great target. ?The gene is small, and encodes an enzyme,? said Dr. Pierce.

The next day, as my talk about the history of gene therapy wound down, I took stock of my audience. Two young women with canes sat in the front row. A few rows back sat Karen Poulakos, also with a cane, whom I?d chatted with earlier.

Karen has Corey?s disease, LCA2, but, at age 63, had been deemed too old for the gene therapy clinical trial two years ago. But things had changed, she?d learned at the meeting, and she just might be eligible for the phase 3 trial coming up. Karen has lived a full life in her world of shadows, barely remembering when she could see better, and she?s now contemplating what it might be like to see again.

As I collected my things, I marveled at the hope radiating from the faces in the room, sighted as well as not. And I thought that this is science at its very best. This is what it is all about, the molecules, the mice, the deciphering of nature?s mechanisms: helping people.

Source: http://rss.sciam.com/click.phdo?i=eaea537b8f67ae32fe8b5a6982030cc1

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Sunday, July 29, 2012

Health weight loss - HEALTH, BEAUTY & FITNESS

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